Recent neuroimaging studies suggest that developmental dyslexia comprises several subtypes with distinct symptom profiles, each associated with characteristic functional and structural abnormalities in the brain. However, how these regional brain-activity abnormalities lead to specific symptoms remains unclear. Moreover, while research using animal models such as mice or rats is effective in studying many brain disorders, it is not suitable for dyslexia because reading and writing are uniquely human skills.

To address this gap, the authors developed a human model of dyslexia, in which key dyslexia-related features are temporarily and safely induced in neurotypical human volunteers. 

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